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| Section | Weight | Objectives |
|---|---|---|
| Embryology, Physiology, Pathophysiology & Systems Management | 54% | - Metabolic and endocrine system - Hematopoietic and immune system - Neurological system - Gastrointestinal system - Renal and genitourinary system - Cardiovascular system - Musculoskeletal and integumentary system - Eyes, ears, nose and throat - Genetics and congenital anomalies - Infectious diseases - Respiratory system |
| General Management | 19% | - Resuscitation and stabilization - Nutrition and growth support - Fluids, electrolytes and acid-base balance - Care coordination and family education - Pain management and sedation - Thermoregulation |
| Pharmacology | 9% | - Common medications and dosages - Safe administration principles - Pharmacokinetics and pharmacodynamics in neonates - Adverse effects and interactions |
| General Assessment | 15% | - Interpretation of diagnostic data - Growth and development assessment - Systematic physical assessment - Perinatal history collection |
| Professional Issues | 3% | - Interprofessional collaboration - Ethics and legal aspects - Quality improvement and patient safety - Scope and standards of practice |
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NEW QUESTION # 89
What condition can occur in the newborn as a result of Rh incompatibility?
Answer: B
Explanation:
Hydrops fetalis occurs as a result of Rh incompatibility between the newborn and the mother. The Rh incompatibility results in a large number of red blood cells in the infant to be destroyed. This causes severe edema. Approximately half of newborns with hydrops fetalis will not survive. This can be prevented if prenatal testing is done on the mother to determine her Rh status.
If she is Rh negative, an injection can be given during the first trimester of her pregnancy to prevent this reaction from occurring.
NEW QUESTION # 90
Identifying and treating congenital hypothyroidism within the first month oflife is especially critical to prevent
Answer: A
Explanation:
Untreated congenital hypothyroidism can result in profound intellectual disability, so diagnosing the disorder and beginning thyroxine treatment within the first month of life is essential. Newborn screening tests in most states mandate checking for hypothyroidism. Infants whose mothers received radioactive iodine treatment for thyroid cancer during pregnancy are especially at risk Signs of congenital hypothyroidism include hypotonia, growth restriction, poor feeding, wide hands with stunted fingers, short extremities, edematous face, and thick protruding tongue.
NEW QUESTION # 91
An elevated serum lactate level in an infant in the Neonatal Intensive Care Unit is associated with
Answer: C
Explanation:
Lactic acid is increased in situations of tissue hypoxia, which can lead to severe metabolic acidosis. This can occur with sepsis, heart failure, shock, and multisystem organ failure. Studies have shown that elevations in serum lactate in the first week of life correlate with an increase in mortality rates. This risk is higher in infants born less than 1000 g.
NEW QUESTION # 92
Which of the following is a defect in chromosome 22?
Answer: B
Explanation:
DiGeorge syndrome occurs when there is a mutation to chromosome 22 during fetal development. It results in an immunodeficiency disorder that affects the thymus gland and the production of T -lymphocyte cells. This results in frequent infections, congenital heart defects, and hypocalcemia. This syndrome also causes characteristic facial features with an underdeveloped chin, heavy eye lids, ears that are rotated back and small upper ear lobes.
NEW QUESTION # 93
A neonate is 40 weeks gestation and nursing well but has onset of jaundice at 36 hours.
Total serum bilirubin is 12 mg/dL At this time, treatment should include
Answer: A
Explanation:
While the neonate's bilirubin is elevated (normal range 3.4-11.5 mg/dL at 1-2 days), only continued observation and jaundice assessment is indicated at this time. Physiologic hyperbilirubinemia is common in newborns and usually benign, resulting from immature hepatic function and increased RBC hemolysis. Infants have larger red blood cells with a shorter life than adults, leading to more RBC destruction and resulting in an increased load of serum bilirubin, which the liver of the newborn cannot handle. Onset is usually within 24-48 hours, peaking in 72 hours for full term or 5 days for preterm infants and declining within a week. Phototherapy is the indicated treatment for total serum bilirubin 218 mg/dL for those at medium risk.
NEW QUESTION # 94
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